Listers,
When we are told we have autoimmune hypothyroidism, does this definitely
indicate the gland will decline in function more and more until it is
totally destroyed?  In that case, it would seem best to ramp up the thyroid
hormone replacement, and just suppress the remaining endogenous function
altogether.  Quality of life on full replacement would then be optimized.  

Or is it possible that the gland could stabilize at a certain functioning
level, autoimmune disease notwithstanding?   If so, a partial replacement
could theoretically top off the endogenous function with good result.

(In my case, when autoantibodies for hashimoto’s were run, they were
negative.  But my endo says an autoimmune process is still probably the
cause, such as vs. rarer proteins, etc.)
 
Right now we are treating the symptoms, raising dose as necessary and not
concerned with how little or how much the gland is suppressed.

Holly

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