PALEOFOOD Archives

Paleolithic Eating Support List

PALEOFOOD@LISTSERV.ICORS.ORG

Options: Use Forum View

Use Monospaced Font
Show Text Part by Default
Show All Mail Headers

Message: [<< First] [< Prev] [Next >] [Last >>]
Topic: [<< First] [< Prev] [Next >] [Last >>]
Author: [<< First] [< Prev] [Next >] [Last >>]

Print Reply
Subject:
From:
Joelle Marble <[log in to unmask]>
Reply To:
Paleolithic Eating Support List <[log in to unmask]>
Date:
Mon, 11 Aug 2003 14:27:04 -0700
Content-Type:
text/plain
Parts/Attachments:
text/plain (34 lines)
> fasting insulin: 3 (normal)
> estrodial: 86 (normal)
> TSH: 2.0 (normal)
> prolactin: 26 (high)
> free T3: low (in the past)
> homocystiene: still out
> CRP: ditto

Here's some info regarding the high levels of prolactin:

Increased prolactin released from the pituitary gland can increase the
brain's dopamine levels (which will then normally feedback to decrease the
prolactin secretion) and increased dopamine can inhibit GnRH release from
the hypothalamus to in turn decrease pituitary FSH and LH secretion. A
decrease in FSH may be the basis for most prolactin associated ovulatory
problems.


There are prolactin receptors on the adrenal glands. The adrenal glands
may respond to increased prolactin by increasing their own androgenic
hormones. The adrenal androgenic hormones are known to interfere with
ovulation.


Prolactin can decrease progesterone production by granulosa cells (the
cells that line ovarian follicles) when grown in culture. If there is a
direct effect of prolactin on granulosa cell progesterone production in
vivo (in a woman's ovaries) then this could also lead to an ovulatory
dysfunction, called a luteal phase defect.


Good Luck
Joelle Marble

ATOM RSS1 RSS2