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>From: smudger <[log in to unmask]>
>Subject: new research on very mad cows disease
>Date sent: Mon, 05 Feb 2001 10:56:43 -0800
>Send reply to: [log in to unmask]
>for your little girl and her lice......
>
>Insecticides Cause Mad Cow Disease Pharmaceutical interests in the
>UK are ignoring new scientific research that shows the insecticide
>used in the UK government's own warble-fly campaigns triggered the UK
>surge of 'Mad Cow' disease. Latest experiments by Cambridge University
>prion specialist, David R. Brown, have shown that manganese bonds with
>prions. Other researchers work shows that prions in the bovine spine
>--along which insecticides are applied-- can be damaged by ICI's
>Phosmet organophosphate(OP) insecticide - causing the disease. British
>scientists have led the current theory that an infectious prion in
>bonemeal fed to cattle causes bovine spongiform disease (BSE).
>Infectious prions are also claimed to cause new variant
>Creutzfeld-Jakob Disease (CJD) in humans -from ingesting beef. But the
>infectious prion theory serves to obscure a tragic chemical poisoning
>scandal behind the majority of BSE cases. The new work proves that the
>prions can bond with manganese in animal feeds or mineral licks. These
>manganese prions cause the neurological degeneration seen in BSE. By a
>similar process, prions in human brains are damaged by lice lotions
>containing organophosphate. This can result in neurological diseases
>like CJD and Alzheimer's -later in life. Many might be surprised to
>hear that organophosphates were developed by Nazi chemists during the
>course World War Two, as a chemical weapon nerve agent. The
>marginalized research has devastating financial implications for ICI.
>It would provide a firm basis for litigants -who could include CJD
>sufferers, farmers across the world and families of the many British
>farmers who committed suicide during this BSE debacle. Scientist
>and organic farmer, Mark Purdey gave evidence to the UK BSE inquiry,
>that warble fly insecticide was the cause of the disease. The scientist
>wheeled out to rubbish Purdy's evidence -Dr. David Ray, later turned
>out to have been receiving funding from the insecticide manufacturer
>ICI. Purdey has been consistently denied even exploratory funding to
>extend his privately supported research. Yet the Purdey/Brown chemical
>poisoning model matches with the epidemiological spread of CJD clusters
>in humans. It also predicts the incidence of BSE-type diseases in
>animals. The accepted infectious model fits neither. The
>pharmaceutical industry is all the more determined to hide the
>chemical source of BSE and CJD, because a spotlight on chemicals would
>expose the role the insecticides in Alzheimer's --another
>neurodegenerative disease- -that might lead to claims which would dwarf
>those from BSE and CJD litigants. In fact, two leading brain
>researchers into CJD and Alzheimer's have died in suspicious
>circumstances in recent years. In the United States, the Environmental
>Protection Agency is already reviewing Phosmet's safety. The Centers
>for Disease Control in the US has recently conducted experiments on
>mice that confirm the organophosphate risk. Not only is the EC beef
>slaughter campaign futile -because BSE disease is mostly
>noninfectious, but unless the underlying chemical cause is addressed,
>BSE will simply reappear from chemical causes. A new warble fly
>campaign is already underway in France using the organophosphate
>insecticide. Of greater concern is that some lotions for scabies and
>head lice are now priming children and adults, for CJD and Alzheimer's
>in later life. Bonding the Prion Cambridge University prion
>biochemist, David R. Brown is dismissive of the science behind the
>infectious model of BSE. He terms it "a very limited amount of science
>by a few assumed- reputable scientists." He insists there is "no
>evidence an infectious agent is present in either meat or milk."
>"Simple tests on udder walls of cows --which could easily detect an
>infectious prion-- have not been done, why I don't understand." A
>number of researchers have found that organophosphate(OP) in systemic
>warble fly insecticide can deform the prion molecule, rendering it
>ineffective at buffering free radical effects in the body. Worse still,
>the prion is then partial to bond with manganese and become a 'rogue'
>prion. A chain reaction whereby rogue prions turn others to rogues
>also, can explain the bovine spongiform disease mechanism. Brown
>showed how prion protein bonds benignly with copper, but lethally with
>manganese. Even natural variations in relative environmental
>availability of manganese versus copper can trigger prion degradation.
> The CJD and BSE symptoms mirror 'manganese madness', an irreversible
>fatal neuro-psychiatric degenerative syndrome that plagued manganese
>miners in the first half of the last century Shining A Light On
>Spongiform Organic dairy farmer and peer-review-published independent
>scientist, Mark Purdey, says the accepted theory of transmission from
>BSE- infected cattle to human CJD -by bonemeal or meat, is dependent on
>a mutant prion that has never been isolated under the scientific
>protocol called Koch's postulates. Purdey's insistence on sticking to
>the letter of this scientific law earned him the condemnation of UK
>officialdom when he first mooted his theory. But Purdey pointed to CJD
>clusters downwind of a British Phosmet production plant to back his
>case. He gave evidence to the UK Government BSE inquiry and was
>supported by Conservative MP, Thessa Gorman. His views were discounted,
>but his subsequent research and the new Cambridge prion work have
>confirmed the alternative theory. Despite this, and the backing of a
>British peer, he is denied even exploratory funding. Why does CJD
>degeneration in humans begin in the retina, and why are CJD disease
>clusters found in high altitude locations? The prion molecule has a
>known natural role as a shock absorber of damaging energy from
>ultraviolet rays and other oxidizing agents. Once this prion defense
>system is rendered ineffective by organophosphates - for example in
>human head lice lotions, these oxidizing effects have an unmediated
>impact on tissues. Eventually, UV radiation damages the retina and
>oxidative stress destroys the brain tissues of CJD patients. This
>theory would expect to find higher CJD incidence in mountain regions
>-where UV radiation levels are elevated. That prediction holds true.
>A similar but accelerated mechanism could be driving BSE. ICI's
>Phosmet organophosphate warble fly insecticide -applied on the backs of
>animals along the spinal column, similarly degrades prions. "Systemic
>versions of the insecticide are designed to make the entire cow
>carcass toxic to warble fly," explains Purdey. "Unfortunately it's
>toxic to prions too -especially those prions located just millimeters
>from the point of application." The damaged prions are then ready to
>react with manganese in animal feed, or manganese sprayed on land or
>in mineral licks -to become the driving force of BSE
>neurodegeneration. Purdey says manganese-tipped prions set off lethal
>chain reactions that neurologically burn through the animal. Chickens
>notoriously excrete most of the supplements fed to them - including
>manganese. And their manganese-rich excreta have been blended into
>cattle feed in the UK. Natural variations in the relative environmental
>availability of copper and manganese can also spur prion degeneration
>says Purdey. From this research, any prudent person would conclude
>there is a significant risk attaching to the use of organophosphate in
>humans. Preparations for head lice and scabies are known to be
>overused in practice and might be priming users for CJ disease. The
>Money Trail Critical scientists like Purdey are unlikely to prevail.
>The pharma industry holds most research purse strings, and would
>hardly energetically explore an avenue of research that could expose
>them to litigation for causing BSE. The official theory is lavishly
>funded, alternative theories rarely, if at all. There are more
>explosive implications to his -and other's latest research. Purdey
>says similar organophosphate-induced protein deformation could also
>underlie Alzheimer's disease. If that were true, the litigation fallout
>would destroy some pharmaceutical giants, and a lot of very influential
>noses would be out of joint. Disturbingly, Purdey and other brain
>researchers seem to have had an undue share of unfortunate accidents.
>Purdey's house was burned down and his lawyer who was working with him
>on Mad Cow Disease was driven off the road by another vehicle and
>subsequently died. The veterinarian on the case also died in a car
>crash -locally reported as: 'Mystery Vet Death Riddle.' Dr. C.
>Bruton, a CJD specialist --who had just produced a paper on a new
>strain of CJD-- was killed in a car crash before his work was announced
>to the public. Purdey speculates that Bruton might have known more than
>what was revealed in his last scientific paper. In 1996, leading
>Alzheimer's researcher Tsunao Saitoh, 46 and his 13 -year-old daughter
>were killed in La Jolla, California, in what a Reuters report described
>as a "very professionally done" shooting. EIONews.com December, 13
>2000 DR. MERCOLA'S COMMENT: You will be hearing more and more of
>Mad Cow Disease and it gradually shifts its way to the US. Most of
>what you read about it ignore the truth of the connection of
>organophosphate pesticides. Mad Cow disease is not common in the US but
>Alzheimer's is and this is a good reason to avoid pesticides. Related
>Articles: Alternatives to Using Pesticides Pesticides May Decrease
>Male Fertility Pesticides Increase Breast Cancer Risk Pesticides May
>Increase Parkinson's Risk [Non-text portions of this message have
>been removed]
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