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Subject:
From:
Kutubo Manneh <[log in to unmask]>
Reply To:
The Gambia and related-issues mailing list <[log in to unmask]>
Date:
Mon, 17 Jul 2000 22:34:18 +0300
Content-Type:
text/plain
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HIV is the causative agent for AIDS,this is irrefutable!!It has been proven
scientifically by using 'Koch's postulates'.Fellow Microbiologists  on the
list will know what i am talking about.Please let us not waste precious time
debating this,as that Great Leader (Mandella) said,we should be thinking how
to solve the pandemic.
  Kind regards
   KUTS
----- Original Message -----
From: sidi sanneh <[log in to unmask]>
To: <[log in to unmask]>
Sent: Monday, July 17, 2000 10:01 PM
Subject: Re: HIV Is Not The Cause of AIDS - Fwd


> Ginny, Buharry, Hamjatta and all those following the debate on the cause
or
> causes of AIDS, in addition to the various the links and articles on both
> sides of the debate, The Economist magazine has an interesting lead story
on
> the issue which I find quite interesting and relevant to the African
debate
> on AIDS. My non-scientist view is that we must approach the problem with
> some pragmatism. The debate must continue by all means, but Africa cannot
> afford to continue trying to re-arrange the deck chairs of the Titanic
while
> the disease takes its toll at such alarming rates.  Please visit The
> Economist web site at www.economist.com/editorial for views which closely
> represent mine. Thanks
> Sidi Sanneh
>
>
> >From: Ginny Quick <[log in to unmask]>
> >Reply-To: The Gambia and related-issues mailing list
> ><[log in to unmask]>
> >To: [log in to unmask]
> >Subject: Re: HIV Is Not The Cause of AIDS - Fwd
> >Date: Mon, 17 Jul 2000 13:37:03 -0500
> >
> >To Buharry and anyone else interested...  There is a website with
articles
> >and information from other "AIDS dissidents", as those who do not follow
> >the
> >mainstream are called.  I stumbled upon the site accidentally from
antoher
> >site I was at.
> >The link is below.  I am not sure what to think, but the information is
> >quite compelling, and to my mind, carries no more or less weight than
what
> >the mainstream info does, considering that it seems we are no closer to
> >solving the AIDS dilemma than we were 15 of 20 or so years ago.
> >Here is the link...
> >http://www.virusmyth.com/
> >Take care,
> >Ginny
> >
> >----- Original Message -----
> >From: "MOMODOU BUHARRY GASSAMA" <[log in to unmask]>
> >To: <[log in to unmask]>
> >Sent: Monday, July 17, 2000 9:51 AM
> >Subject: HIV Is Not The Cause of AIDS - Fwd
> >
> >
> >Hi Hamjatta!
> >                   The articles you forwarded on AIDS are enlightening.
The
> >South African president was talking about AIDS from the Duesberg
> >perspective
> >which has caused such an uproar. I'll forward some articles dealing with
> >this and the conspiracy theory that you talked about in your other
posting.
> >Have a good day.
> >
> >Buharry.
> >____________________________________________________________________
> >
> >
> >HIV Is Not the Cause of AIDS
> >By Peter H. Duesberg
> >
> >Science, Vol. 241, pp. 514-517, July 29, 1988.
> >
> >
> >
> >
> >
> >HIV Is Not the Cause of AIDS
> >Human immunodeficiency virus (HIV) is not the cause of AIDS because it
> >fails
> >to meet the postulates of Koch and Henle, as well as six cardinal rules
of
> >virology.
> >     1) HIV is in violation of Koch's first postulate because it is not
> >possible to detect free virus (1, 2), provirus (3-5), or viral RNA (4, 6,
> >7)
> >in all cases of AIDS. Indeed, the Centers for Disease Control (CDC) has
> >established guidelines to diagnose AIDS when all laboratory evidence for
> >HIV
> >is negative (8).
> >     2) In violation of Koch's second postulate, HIV cannot be isolated
> >from
> >20 to 50% of AIDS cases (1, 9-11). Moreover, "isolation" is very
indirect.
> >It depends on activating dormant provirus in millions of susceptible
cells
> >propagated in vitro away from the suppressive immune system of the host.
> >     3) In violation of Koch's third postulate, pure HIV does not
reproduce
> >AIDS when inoculated into chimpanzees or accidentally into healthy humans
> >(9, 12, 13).
> >     4) In contrast to all pathogenic viruses that cause degenerative
> >diseases, HIV is not biochemically active in the disease syndrome it is
> >named for (14). It actively infects only 1 in 104 to > 105 T cells (4, 6,
> >7,
> >15). Under these conditions, HIV cannot account for the loss of T cells,
> >the
> >hallmark of AIDS, even if all infected cells died. This is because during
> >the 2 days it takes HIV to replicate, the body regenerates about 5% of
its
> >T
> >cells (16), more than enough to compensate for losses due to HIV.
> >     5) It is paradoxical that HIV is said to cause AIDS only after the
> >onset
> >of antiviral immunity, detected by a positive "AIDS test," because all
> >other
> >viruses are most pathogenic before immunity. The immunity against HIV is
so
> >effective that free virus is undetectable (see point 1), which is why HIV
> >is
> >so hard to transmit (9, 12, 13). The virus would be a plausible cause of
> >AIDS if it were reactivated after an asymptomatic latency, like herpes
> >viruses. However, HIV remains inactive during AIDS. Thus the "AIDS test"
> >identifies effective natural vaccination, the ultimate protection against
> >viral disease.
> >     6) The long and highly variable intervals between the onset of
> >antiviral
> >immunity and AIDS, averaging 8 years, are bizarre for a virus that
> >replicates within 1 to 2 days in tissue culture and induces antiviral
> >immunity within 1 to 2 months after an acute infection (9, 17). Since all
> >genes of HIV are active during replication, AIDS should occur early when
> >HIV
> >is active, not later when it is dormant. Indeed, HIV can cause a
> >mononucleosis-like disease during the acute infection, perhaps its only
> >pathogenic potential (9, 17).
> >     7) Retroviruses are typically not cytocidal. On the contrary, they
> >often
> >promote cell growth. Therefore, they were long considered the most
> >plausible
> >viral carcinogens (9). Yet HIV, a retrovirus, is said to behave like a
> >cytocidal virus, causing degenerative disease killing billions of T cells
> >(15, 18). This is said even though T cells grown in culture, which
produce
> >much more virus than has ever been observed in AIDS patients, continue to
> >divide (9, 10, 18).
> >     8) It is paradoxical for a virus to have a country-specific host
range
> >and a risk group-specific pathology. In the United States, 92% of AIDS
> >patients are male (19), but in Africa AIDS is equally distributed between
> >the sexes, although the virus is thought to have existed in Africa not
much
> >longer than in the United States (20). In the United States, the virus is
> >said to cause Kaposi's sarcoma only in homosexuals, mostly Pneumocystis
> >pneumonia in hemophiliacs, and frequently cytomegalovirus disease in
> >children (21). In Africa the same virus is thought to cause slim disease,
> >fever, and diarrhea almost exclusively (22, 23).
> >     9) It is now claimed that at least two viruses, HIV-1 and HIV-2, are
> >capable of causing AIDS, which allegedly first appeared on this planet
only
> >a few years ago (20). HIV-1 and HIV-2 differ about 60% in their nucleic
> >acid
> >sequences (24). Since viruses are products of gradual evolution, the
> >proposition that within a few years two viruses capable of causing AIDS
> >could have evolved is highly improbable (25).
> >
> >References and Notes:
> >   1.. J. Albert et al., J. Med. Virol. 23, 67 (1987).
> >   2.. L.A. Falk, D. Paul, A. Landay, H. Kessler, N. Engl. J. Med. 316,
> >1547
> >(1987).
> >   3.. G.M. Shaw et al., Science 226, 1165 (1984).
> >   4.. D. Richman, J. McCutchan, S. Spector, J. Infect Dis. 156, 823
> >(1987).
> >   5.. C.-Y. Ou et al., Science 239, 295 (1988).
> >   6.. M.E. Harper, L.M. Marselle, R.C. Gallo, F. Wong-Staal, Proc. Natl.
> >Acad. Sci. U.S.A. 83, 772 (1986).
> >   7.. A. Ranki et al., Lancet ii, 589 (1987).
> >   8.. Centers for Disease Control, J. Am. Med. Assoc. 258, 1143 (1987).
> >   9.. P.H. Duesberg, Cancer Res. 47, 1199 (1987).
> >   10.. H. von Briesen et al., J. Med. Virol. 23, 51 (1987).
> >   11.. D. Gallo, J. Kimpton, P. Dailey, J. Clin. Microbiol. 25, 1291
> >(1987).
> >   12.. J.W. Curran et al., Science 239, 610 (1988).
> >   13.. G.H. Friedland and R.S. Klein, N. Engl. J. Med. 317, 1125 (1987).
> >   14.. J. Coffin et al., Science 232, 697 (1986).
> >   15.. A. Fauci, ibid. 239, 617 (1988).
> >   16.. J. Sprent, in B and T Cells in Immune Recognition, F. Loor and
G.E.
> >Roelants, Eds. (Wiley, New York, 1977), pp. 59-82.
> >   17.. H.A. Kessler, J. Am. Med. Assoc. 258, 1196 (1987).
> >   18.. R.C. Gallo, Sci. Am. 256 (No. 1), 47 (1987).
> >   19.. Centers for Disease Control, AIDS Weekly Surveill. Rep., 18 April
> >1988.
> >   20.. R. Baum, "AIDS: The molecular biology," Chem. Eng. News (23
> >November
> >1987), pp. 14-26.
> >   21.. R.M. Selik, E.T. Starcher, J.W. Curran, AIDS 1, 175 (1987).
> >   22.. R. Colebunders et al., Lancet i, 492 (1987).
> >   23.. K.J. Pallangyo et al., ibid. ii, 972 (1987).
> >   24.. F. Clavel et al., Nature 324, 691 (1986).
> >   25.. J. Sonnabend, in New York Native (9 May 1988), p. 19.
> >
>
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