To Buharry and anyone else interested... There is a website with articles
and information from other "AIDS dissidents", as those who do not follow the
mainstream are called. I stumbled upon the site accidentally from antoher
site I was at.
The link is below. I am not sure what to think, but the information is
quite compelling, and to my mind, carries no more or less weight than what
the mainstream info does, considering that it seems we are no closer to
solving the AIDS dilemma than we were 15 of 20 or so years ago.
Here is the link...
http://www.virusmyth.com/
Take care,
Ginny
----- Original Message -----
From: "MOMODOU BUHARRY GASSAMA" <[log in to unmask]>
To: <[log in to unmask]>
Sent: Monday, July 17, 2000 9:51 AM
Subject: HIV Is Not The Cause of AIDS - Fwd
Hi Hamjatta!
The articles you forwarded on AIDS are enlightening. The
South African president was talking about AIDS from the Duesberg perspective
which has caused such an uproar. I'll forward some articles dealing with
this and the conspiracy theory that you talked about in your other posting.
Have a good day.
Buharry.
____________________________________________________________________
HIV Is Not the Cause of AIDS
By Peter H. Duesberg
Science, Vol. 241, pp. 514-517, July 29, 1988.
HIV Is Not the Cause of AIDS
Human immunodeficiency virus (HIV) is not the cause of AIDS because it fails
to meet the postulates of Koch and Henle, as well as six cardinal rules of
virology.
1) HIV is in violation of Koch's first postulate because it is not
possible to detect free virus (1, 2), provirus (3-5), or viral RNA (4, 6, 7)
in all cases of AIDS. Indeed, the Centers for Disease Control (CDC) has
established guidelines to diagnose AIDS when all laboratory evidence for HIV
is negative (8).
2) In violation of Koch's second postulate, HIV cannot be isolated from
20 to 50% of AIDS cases (1, 9-11). Moreover, "isolation" is very indirect.
It depends on activating dormant provirus in millions of susceptible cells
propagated in vitro away from the suppressive immune system of the host.
3) In violation of Koch's third postulate, pure HIV does not reproduce
AIDS when inoculated into chimpanzees or accidentally into healthy humans
(9, 12, 13).
4) In contrast to all pathogenic viruses that cause degenerative
diseases, HIV is not biochemically active in the disease syndrome it is
named for (14). It actively infects only 1 in 104 to > 105 T cells (4, 6, 7,
15). Under these conditions, HIV cannot account for the loss of T cells, the
hallmark of AIDS, even if all infected cells died. This is because during
the 2 days it takes HIV to replicate, the body regenerates about 5% of its T
cells (16), more than enough to compensate for losses due to HIV.
5) It is paradoxical that HIV is said to cause AIDS only after the onset
of antiviral immunity, detected by a positive "AIDS test," because all other
viruses are most pathogenic before immunity. The immunity against HIV is so
effective that free virus is undetectable (see point 1), which is why HIV is
so hard to transmit (9, 12, 13). The virus would be a plausible cause of
AIDS if it were reactivated after an asymptomatic latency, like herpes
viruses. However, HIV remains inactive during AIDS. Thus the "AIDS test"
identifies effective natural vaccination, the ultimate protection against
viral disease.
6) The long and highly variable intervals between the onset of antiviral
immunity and AIDS, averaging 8 years, are bizarre for a virus that
replicates within 1 to 2 days in tissue culture and induces antiviral
immunity within 1 to 2 months after an acute infection (9, 17). Since all
genes of HIV are active during replication, AIDS should occur early when HIV
is active, not later when it is dormant. Indeed, HIV can cause a
mononucleosis-like disease during the acute infection, perhaps its only
pathogenic potential (9, 17).
7) Retroviruses are typically not cytocidal. On the contrary, they often
promote cell growth. Therefore, they were long considered the most plausible
viral carcinogens (9). Yet HIV, a retrovirus, is said to behave like a
cytocidal virus, causing degenerative disease killing billions of T cells
(15, 18). This is said even though T cells grown in culture, which produce
much more virus than has ever been observed in AIDS patients, continue to
divide (9, 10, 18).
8) It is paradoxical for a virus to have a country-specific host range
and a risk group-specific pathology. In the United States, 92% of AIDS
patients are male (19), but in Africa AIDS is equally distributed between
the sexes, although the virus is thought to have existed in Africa not much
longer than in the United States (20). In the United States, the virus is
said to cause Kaposi's sarcoma only in homosexuals, mostly Pneumocystis
pneumonia in hemophiliacs, and frequently cytomegalovirus disease in
children (21). In Africa the same virus is thought to cause slim disease,
fever, and diarrhea almost exclusively (22, 23).
9) It is now claimed that at least two viruses, HIV-1 and HIV-2, are
capable of causing AIDS, which allegedly first appeared on this planet only
a few years ago (20). HIV-1 and HIV-2 differ about 60% in their nucleic acid
sequences (24). Since viruses are products of gradual evolution, the
proposition that within a few years two viruses capable of causing AIDS
could have evolved is highly improbable (25).
References and Notes:
1.. J. Albert et al., J. Med. Virol. 23, 67 (1987).
2.. L.A. Falk, D. Paul, A. Landay, H. Kessler, N. Engl. J. Med. 316, 1547
(1987).
3.. G.M. Shaw et al., Science 226, 1165 (1984).
4.. D. Richman, J. McCutchan, S. Spector, J. Infect Dis. 156, 823 (1987).
5.. C.-Y. Ou et al., Science 239, 295 (1988).
6.. M.E. Harper, L.M. Marselle, R.C. Gallo, F. Wong-Staal, Proc. Natl.
Acad. Sci. U.S.A. 83, 772 (1986).
7.. A. Ranki et al., Lancet ii, 589 (1987).
8.. Centers for Disease Control, J. Am. Med. Assoc. 258, 1143 (1987).
9.. P.H. Duesberg, Cancer Res. 47, 1199 (1987).
10.. H. von Briesen et al., J. Med. Virol. 23, 51 (1987).
11.. D. Gallo, J. Kimpton, P. Dailey, J. Clin. Microbiol. 25, 1291 (1987).
12.. J.W. Curran et al., Science 239, 610 (1988).
13.. G.H. Friedland and R.S. Klein, N. Engl. J. Med. 317, 1125 (1987).
14.. J. Coffin et al., Science 232, 697 (1986).
15.. A. Fauci, ibid. 239, 617 (1988).
16.. J. Sprent, in B and T Cells in Immune Recognition, F. Loor and G.E.
Roelants, Eds. (Wiley, New York, 1977), pp. 59-82.
17.. H.A. Kessler, J. Am. Med. Assoc. 258, 1196 (1987).
18.. R.C. Gallo, Sci. Am. 256 (No. 1), 47 (1987).
19.. Centers for Disease Control, AIDS Weekly Surveill. Rep., 18 April
1988.
20.. R. Baum, "AIDS: The molecular biology," Chem. Eng. News (23 November
1987), pp. 14-26.
21.. R.M. Selik, E.T. Starcher, J.W. Curran, AIDS 1, 175 (1987).
22.. R. Colebunders et al., Lancet i, 492 (1987).
23.. K.J. Pallangyo et al., ibid. ii, 972 (1987).
24.. F. Clavel et al., Nature 324, 691 (1986).
25.. J. Sonnabend, in New York Native (9 May 1988), p. 19.
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