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Mon, 6 Feb 2006 14:45:05 EST |
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Because of friends and clients with biochemical conditions requiring lithium
treatment and also with severe hypothyroid symptoms, I have been concerned
for years about how they are treated (or mis-treated). The posting about the
effect of lithium on the cells' ability to take in and use T3 is significant
and seems to answer some of my questions.
If the lithium helps you otherwise, don't quit it.
Are your doctors willing to learn from your previous doctor?
T4 is the 'storage' form, ready to be converted. T3 is the form that gets
into the cells if it can get in. Reverse T3 is the form that is not usable,
but it gets counted with the normal T3 in the usual blood tests that only
count iodine atoms on the molecule.
I have wondered about the role of cortisol and the role of the pituitary in
those biochemical conditions.
Cortisol levels fluctuate in the day normally, but what happens in people
with biochemical conditions that need lithium for treatment? Apparently
cortisol levels affect whether T4 converts to T3 at all and whether it converts to
T3 or Reverse T3 (the unusable form). Is it also a factor in whether normal
T3 gets into the cellular receptor sites? Anyway, the unused and unusable
thyroid hormones, if they stay in the body, lower TSH even if the person is
hypothyroid at the cellular level and has all the symptoms of being hypothyroid.
I also wonder if cells that have been starved for T3 are like human victims
of starvation. When starved and emaciated humans first get real food, they
can only take in a little at a time (starting with broth) until 'ready' to eat
normally. I developed that theory because of my experiences.
Peg
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