<<Disclaimer: Verify this information before applying it to your situation.>>
Included here are suggestions or comments about rheumatoid arthritis:
Some help for negotiating web pages: (sorry, I meant to include this with
the web sites)
If you are using Internet Explorer, click on one of the above links and
wait for the website to display. Then click on the word 'Edit' on the menu
bar at the very top of your screen. This is the menu bar that
starts "File" "Edit" "View", etc.
When the Edit submenu displays, click on the item called "Find (on this
page)". A little box will pop up and you can type in a word or phrase
(rheumatoid, for example), click the Find Next box and it will take you to
the section that references the word you entered. This will help you find
relevant information without having to read the entire article, as some
references to RA and diabetes are pretty far down in several articles.
Other Suggestions:
Check out the archives; there's quite a bit in there about RA and celiac
disease. Most people seem to report that when they start eating gluten
free, their pain is relieved, at least significantly if not completely.
I think you will find yoga helpful (gentle yoga).
Exactly how low acidity is related to these conditions is not well
understood. "Leaky gut" is involved. I could guess that low acidity might
lead to secondary conditions which may in turn trigger a variety of
autoimmune diseases. You may benefit from a hydrochloric acid supplement,
betaine hydrochloride (HCl). There are "self-tests" described on-line or
you may consider having the Heidelberg capsule test for stomach acid
levels.
Contact Gluten Intolerance Group™ (GIG™) in Seattle, WA for information.
Gluten Intolerance Group™
15110 10th Ave SW, Suite A
Seattle, WA 98166-1820
Telephone: 206-246-6652 Fax: 206-246-6531
Website: www.gluten.net Email: [log in to unmask]
Executive Director: Cynthia Kupper, CRD (Certified Registered Dietitian)
(and she has Celiac Disease) is very willing to work with anyone that
calls. Cynthia also has credentials as a former CDE (Certified Diabetes
Educator).
we are very susceptible to 'leaky gut'---look up this term, you'll find it--
--look up 'night shades' these are the veggies with solanine(sp)--tomatoes-
peppers-eggplant-potatos(not the sweets and yams and such)--cigarettes---I
believe these are all----but, off course these veges are incorporated in
much of our foods-----if you are using a lot of citrus—anything with citric
acids too-----notice aches or swelling---work on these ideas
For those of you interested in perusing the current scientific literature
(peer-reviewed science and medical journals), a useful website is Pubmed,
which can be reached at the following link:
http://www.ncbi.nlm.nih.gov/entrez/query.fcgi
This website allows you to search based on author, subject, and several
other criteria. It will then link you to abstracts from various journals,
which you can sort by publication date, journal, author, etc. Some of the
articles can be downloaded full-text from Pubmed. For others, you will
have to either purchase the article online or get it from a local
university or public library.
However, for those of you curious about issues like oats and gluten
content, current Celiac research, and so forth, this is an excellent
resource. I recommend journals such as Science, Nature, The Lancet, or The
New England Journal of Medicine for more "broad" scientific and clinical
reviews. There are also several medical journals that focus on
gasteronterology research that have published excellent articles on Celiac.
I've been reading many medical articles on these links and went to a
national Digestive Disease conference and listened to all the Celiac
Related presentations of Dr. Alessio Fasano of U. of Maryland, spoke
personally with Dr. Joseph Murray of Mayo Clinic, Dr. Kelly (Refractory
Sprue Specialist), and Dr. MacDonald of I think of UK. Have you had HLA
typing? This is a very important key because of the following
quote, "Approximately 40% of the genetic susceptibility in
Caucasians is explained by the high risk alleles > HLA DR3-DQ2 and HLA DR4-
DQ8. The HLA DR2 locus confers dominant protection. One of these
susceptibility alleles HLA DR3-DQ2 (A1*501, B1*0201) has interesting
associations. It is associated with type 1 diabetes in congenital rubella,
coeliac disease and selective IgA deficiency; the latter two conditions
show high antibodies to cows milk protein. It has also been associated with
higher levels of antibodies to GAD in at-risk subjects for type 1 diabetes
and with increased immunity to cows milk protein in diabetic patients and
controls. It seems possible that this haplotype and/or other haplotypes
predispose an individual to sensitization to dietary and viral antigens and
thereby increase the risk of autoimmune disease. It is also possible that
an alteration in gut mucosal immune function in genetically susceptible
individuals, the gut mucosa being the major immunoregulatory barrier in the
infant, underlies an effect of dietary or viral proteins on islet
autoimmunity in early life" (Journal of Paediatrics > and Child Health,
Vol. 37, Issue 3, Page 218, 6/01."
This is from Pediatrics, Vol. 109, No. 5, 5/02, "Diabetes and CD are both
autoimmune disorders, sharing the same high-risk HLA DQ2 genotype. One
third of type 1 diabetes patients with the CD-associated HLA DQ2 genotype
tested serologically positive for CD compared with <2% of patients lacking
DQ2. Other autoimmune diseases associated with CD and type 1 diabetes
include autoimmune thyroiditis, pernicious anemia, Sjogren syndrome,
Addison's disease, alopecia areata, and rheumatoid arthritis. The reasons
for these associations may be that CD and these other disorders share a
similar autoimmune pathogenic mechanism, or that the same gene is
responsible for a proportion of these disorders. It is possible that
chronic lymphocyte stimulation in the intestine in CD could result in an
increase in autoantibody production and therefore stimulate the development
of other autoimmune disorders. Older, untreated CD patients have a higher
prevalence of autoantibodies that younger patients, suggesting that
duration of gluten exposure increases the risk of developing
autoantibodies. In some individuals with rehematoid arthritis and
pericarditis, the conditions disappeared when the patients followed gluten-
free diets. Although type 1 diabetes is occasionally identified in
previously diagnosed individuals with CD, in most cases CD antibodies are
present at the time of or after diagnosis of type 1 diabetes. Diabetic
autoantibodies generally precede the onset of clinical diabetes by an
average of 3 years. .... There is however, no evidence that treating CD
with a gluten-free diet prevents type 1 diabetes."
* Please carefully compose your subject lines in all posts *
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